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GcMAF, also called DBP-MAF, is really a macrophage activating factor (MAF) 

created by enzymatic crystallization of DBP (abbreviation of vitamin D binding protein) in humans; here DBP is really a glycoprotein with about 58,000 The molecular weight of Dalton, owned by the albumin group, is abundantly present in the blood, wherein the DBP is with the vitamin D derivative and transported in the blood. The DBP-blood concentration is approximately 300-600 micrograms per milliliter. The sugar side chain comprises N-acetyl galactosamine, which is formed by crosslinking of galactose and sialic acid. After immunoactivation, the sugar chain of the GC protein is hydrolyzed by the inducible cell membrane β-galactosidase of B cells to make a macrophage pre-activating factor. The activating factor protein was then hydrolyzed via the cell membrane sialidase (neuraminidase) of activated T cells to the final GcMAF with N-acetaminogalactosamine (GaINAc) as the residual residual sugar. This N-acetaminogalactose (GaINAc) is the main element to the benefits of GcMAF.

The enzyme “Nagalase” (alpha-N-Acetylgalactosaminidase) is really a lysosomal enzyme in the liver that cleaves between GaINAc and the protein’s threonine or serine residues. Bonding. The Nagalase enzyme cleaves the sugar side chain of the Gc protein, gcmaf cancer thereby losing its precursor activity and not converting to the active GcMAF. Furthermore, Nagalase also inactivates active GcMAF by saccharide cleavage.

Macrophages play an integral role in immune defense and enter the blood with young, immature monocytes from the bone marrow. In the blood, it differentiates into dendritic cells or, after activation, enters the tissue, where it matures into macrophages of resident tissues, and then acts as a Kufu-type stellate cell (liver), glial cells (neural) System), alveolar macrophages (lung), osteoblasts (bone), or Langham’s giant cells (skin).

This GcMAF clearly has great potential for activation of monocytes and macrophages. Even the lowest concentration of antibacterial products that stimulate oxygen free radicals, the “oxidative burst” of monocytes is up to 50 times. The phagocytic activity and HLA molecular expression increased by way of a factor of 100, and its antigen presentation can grow exponentially. By this enhanced effect, it’s possible to more effectively combat the pathogens that were previously limited to immunodeficiency. This activates an immune response to an unrecognizable antigen. It can decompose and kill tumor cells which were immunologically recognized.

GCMAF YOGURT

The Gcmaf colostrum fermentation should really be enriched with vitamin D3. This vitamin D3 is advantageously put in one of many three binding sites of macrophage activating factor (MAF) and the MAF is thus activated.

The fermentation needs to be enriched with colostrum. The colostrum is employed being an aid in initiating the fermentation. Furthermore, the MAF has three binding sites activated by vitamin D3, colostrum and oleic acid during fermentation.

The fermentation will soon be full of oleic acid since it is containd in the milk and colostrum. An edge of the oleic acid is that additionally, it may activate MAF by binding to one of many three binding sites of MAF. For this reason full fat cows milk should continually be used when coming up with gcmaf yogurt.

Important nutrients are offered in a sufficient form for the bacteriological culture in the nutrient medium.

The GcMAF is created by fermentation predicated on a bacterial culture (organism) in that the culture of the bacteria could be obtained. The fermentation is carried out in milk (similar to the preparation of yogurt). The microorganisms useful for fermentation are delivered in powder form. Furthermore, vitamin D3, colostrum and oleic acid are supplied separately.

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